Most viruses die when an animal is killed and body temperature drops and all viruses die when meat is cooked.
Even the feared HIV dies when body temperature drops to just 32.
1) Virus is not a living being. It could be inactivated or destroyed, but it can't die.
2) Usage of the word "even" is inappropriate here. A stability of the virus in the environment is determined by the composition of its capsid and envelope, and has absolutely no connection with its pathogenicity or virulence. Virus could be unsustainable and unable to "survive" in the environment but it wouldn't prevent it from being extremely pathogenic/virulent. And vice versa, virus could be extremely stable but harmless.
Some examples:
Retroviridae(HIV/FIV/SIV/SHIV, HTLV-1, HTLV-2) or Flaviviridae (Hepatitis B, C) are using Lipid bilayer envelope. Such type of envelopes is unsustainable in the environment and decays quickly enough, destruction process could be accelerated by drying, light or oxigen exposition. These types of envelope are also vulnerable to formaline, organic solvents like gasoline or alcohol and majority of disinfectants.
Poxviridae (Variola Major and Variola Minor), Filoviridae (Ebola and Marburg viruces) are using lipid-protein envelope. Sych types of envelope could be extreme stable in aggressive environment. Variola virus is able to withstand drying in the sunlight for many weeks. Poxviridae envelope consist of three layers, such construction is able to protect viral genetic material even against formaline, phenol or diethyl ether exposition. Seen in this light, it's pretty understandable why smallpox was such a big problem.
By the way, speaking about virulence... It's interesting that lethality rate of Flaviviridae family members is slowly decreasing in case of consequent infection. First transmission of "wild" strain from animal to human results with almost 100% lethality, but next transmissions (from one human to another and so on) produce strains with lower lethality rate. And after 10 consequent transmissions we're getting a strain with "only" 60% lethality rate, for example. It seems that virus adaptation process went similar in the wild nature. A sequence of transmissions from one monkey to another eventually created an adapted strain, which is harmless for them.