Food Totalitarianism:
http://www.bibliotecapleyades.net/sociopolitica/esp_sociopol_depopu36.htmCodex Alimentarius Commission - A Threat to Humankind
There is an entire industry with an innate economic interest to obstruct, suppress and discredit any information about the eradication of diseases. The pharmaceutical industry makes over one trillion dollars from selling drugs for ongoing diseases. These drugs may relieve symptoms, but they do not cure.
We have to realize that the mission of this industry is to make money from ongoing diseases.
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8. Millions of people and patients around the world are defrauded twice: A major portion of their income is used up to finance the exploding profits of the pharmaceutical industry. In return, they are offered a medicine that does not even cure.
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Codex is not an easy subject to get to grips with. With over 20 committees meeting on an annual basis, and published reports comprising a total of over 1,400 pages in 2005 alone, most people are blissfully unaware of the extent to which its activities affect their health.
Read on to discover the bigger picture behind the Codex Alimentarius Commission's support for the "business with disease".
What is Codex?
The World Trade Organization uses Codex Guidelines and Standards as the benchmark in the adjudication of international trade disputes involving foods. It's headquarters, above, are located in Geneva, Switzerland.
The Codex Alimentarius Commission (Codex) is the main global body that makes proposals to, and is consulted by, the Directors-General of the World Health Organization (WHO) and the Food and Agriculture Organization of the United Nations (FAO) on all matters pertaining to the implementation of the Joint FAO/WHO Food Standards Programme.
Established in 1963, the Commission's main purposes are stated in its Procedural Manual as being:
* protecting the health of consumers
* ensuring fair practices in the food trade
* promoting coordination of all food standards work undertaken by international governmental and non-governmental organizations
Unfortunately however, and as we shall see, its activities do not protect the health of consumers and the international food trade is anything but fair.
At the time of writing, the Commission presides over a total of 27 active subsidiary committees and ad hoc intergovernmental task forces, the main functions of which revolve around the drafting of standards, guidelines and other related texts for foods, including food supplements.
Once completed these texts are presented to the Commission for final approval and adoption as new global standards.
How does Codex affect you and your health?
Codex standards and guidelines now exist for virtually all foods.
Whilst the adoption by countries of the various standards and guidelines developed by Codex is theoretically optional, the creation of the World Trade Organization (WTO) on 1 January 1995 essentially changed their international status, in that they are now increasingly used by the WTO as the benchmark in the adjudication of international trade disputes involving foods.
https://en.wikipedia.org/wiki/Raw_milkFrance
Raw milk and especially raw milk cheeses are considered the standard for high quality dairy products. Many French cuisine traditionalists consider pasteurized cheeses almost a sacrilege. Many traditional French cheeses have solely been made from raw milk for hundreds of years.
French Roquefort, a famous blue cheese, which is required by European law to be made from raw sheep's milk.
Canada
The sale of raw milk directly to consumers is prohibited in Canada under the Food and Drug Regulations since 1991.
No person shall sell the normal lacteal secretion obtained from the mammary gland of the cow, genus Bos, or of any other animal, or sell a dairy product made with any such secretion, unless the secretion or dairy product has been pasteurized by being held at a temperature and for a period that ensure the reduction of the alkaline phosphatase activity so as to meet the tolerances specified in official method MFO-3, Determination of Phosphatase Activity in Dairy Products, dated November 30, 1981.
— [28], Section B.08.002.2 (1)
Australia
The sale of raw milk for drinking purposes is illegal in all states and territories in Australia, as is all raw milk cheese. This has been circumvented somewhat by selling raw milk as bath milk.[45] An exception to the cheese rule has been made recently for two Roquefort cheeses. There is some indication of share owning cows, allowing the "owners" to consume the raw milk,[47] but also evidence that the government is trying to close this loophole.
In 2014, after a 3 year old died of haemolytic uraemic syndrome and 4 other children became seriously ill, the Victorian government created new regulations which require producers to treat raw milk to reduce pathogens, or to make the product unpalatable to taste, such as making it bitter.
United States
Main article: United States raw milk debate
In the early 20th century many states allowed the sale of raw milk that was certified by a "medical milk commission", effectively allowing an alternative of extra inspection for pasteurization. Now most states impose restrictions on raw milk suppliers due to concerns about safety. Twenty-eight U.S. states allow sales of raw milk. Cow shares can be found, and raw milk purchased for animal consumption in many states where retail for human consumption is prohibited. The sale of raw milk cheese is permitted as long as the cheese has been aged for 60 days or more.
The FDA reports that, in 2002, consuming partially heated raw milk and raw milk products caused 200 Americans to become ill in some manner.
Many governmental officials and the majority of public health organizations hold to the need for pasteurization. Before pasteurization, many dairies, especially in cities, fed their cattle on low-quality food, and their milk was rife with dangerous bacteria. Pasteurizing it was the only way to make it safely drinkable. As pasteurization has been standard for many years, it is now widely assumed that raw milk is dangerous. The Cornell University Food Science Department has compiled data indicating that pathogenic microorganisms are present in between 0.87% and 12.6% of raw milk samples.
Proponents of raw milk (in the U.S.) advance two basic arguments for unpasteurized milk. They state that pasteurization destroys or damages some of the milk's nutrients, and that while pasteurization may kill dangerous bacteria, it also kills off good bacteria that raw milk supporters have stated to have health benefits. The United States Food and Drug administration has stated that this is false, and that pasteurizing milk does not destroy any of its nutritive value.
Proponents also invoke the benefits of direct-marketing when promoting the sale of raw milk. The ability of the farmer to eliminate the middle-man and sell directly to the consumer allows for greater profitability. Many manufacturers sell small-scale pasteurization equipment, thereby allowing farmers to both bypass the milk processors and sell pasteurized milk directly to the consumer. Additionally, some small U.S. dairies are now beginning to adopt low-temperature vat pasteurization. Advocates of low-temperature vat pasteurization note that it produces a product similar to raw milk in composition and is not homogenized.
http://www.npr.org/sections/thesalt/2015/02/20/387558373/why-some-states-want-to-legalize-raw-milk-salesBut NASDA's Ehart suggests some states may not be legalizing raw milk sales to condone it. Rather, he says legalization may give public health agencies the power to regulate a market that might otherwise exist underground.
http://www.naturalnews.com/035716_raw_milk_legal_directory.htmlA few states allow retail sales of raw milk. In those states, the FDA, USDA, and local health officials tend to harass outspoken raw milk dairy farmers. It's difficult to believe that California is one of the few states allowing raw milk sales. The recent Rawsome Foods raid certainly contradicts that legality (
http://www.naturalnews.com/033225_Rawesome_Foods_photos.html).
http://www.realrawmilkfacts.com/raw-milk-regulationsThe Code of Federal Regulations (21 CFR Sec. 1240.61) mandates pasteurization for all milk and milk products in final package form intended for direct human consumption.
"No person shall cause to be delivered into interstate commerce or shall sell, otherwise distribute, or hold for sale or other distribution after shipment in interstate commerce any milk or milk product in final package form for direct human consumption unless the product has been pasteurized or is made from dairy ingredients (milk or milk products) that have all been pasteurized..."
http://www.foodrenegade.com/healthy-cheese-what-to-buy/What to Buy
Basically, we’re trying to find REAL cheese — cheese that’s as traditional and natural as possible, the kind of cheese your ancestors have been eating for thousands of years.
BEST CHOICE: Raw cheeses from grass-fed cows producing milk high in A2 beta casein and relatively low in A1 beta casein — that means milk from Jerseys, Guernseys, and other traditional cattle breeds rather than newer Holsteins. Raw goat’s milk, sheep’s milk, and yak’s milk only contains A2 beta casein and arguably makes better cheeses than cow’s milk anyway.
Finding these in your grocery store can be tough, but it is possible to find them in many of the “deli” sections. In other words, they won’t be on the same aisle with sliced sandwich cheeses and bagged pre-shredded cheese (which contains wood pulp!). Imported European cheeses are a great place to start. Europeans don’t treat their dairy cows with growth hormones, and they also know that the best tasting cheeses are the ones coming from cows eating lush green grass. You can tell if a cheese is made with raw milk by reading the ingredients label.
Unfortunately, U.S. import laws don’t allow us to import many of the softer or mildly aged raw cheeses, instead requiring that raw cheese be aged for at least 60 days before entering our country. So, you won’t find raw chevre, feta, blue cheese, or cream cheese varieties at your grocery store. For those, you’ll need to go to your farmer’s market, a local artisan cheese shop, or purchase them online.
http://gutcritters.com/small-intestinal-bacterial-overgrowth-part-six-dietary-causes-of-impaired-intestinal-movement-and-inflammation/A1 Beta-casein
Just as gluten opioids slow digestive function, so too A1 beta casein. However, whole pastured dairy is chock full of fat-soluble vitamins, minerals, conjugated linoleic acid, anti-microbial agents and whey protein, all of which have beneficial effects on human health.
Many observational studies that show an association between dairy consumption and poor health outcomes fail to account for gluten consumption. Dairy and wheat often go together: cereal and milk, milk and cookies, ice cream and cake, butter and toast, cheese and crackers, etc. So is it the dairy or the wheat? My bet is on the wheat but we don’t really know as there are no clinically controlled trials that have studied this.
If consuming A1 casein dairy slows things down for you and you’re battling SIBO or trying to heal your leaky gut, you may want to cut it out of your diet or try switching to A2 casein dairy sources until you get things under control. Many feel better cutting dairy completely out of their diets for good while others feel better when they eat it. I certainly credit whole-pastured, A2 casein Jersey milk for part of my recovery from a damaged gut wall caused by SIBO. But that’s my experience not yours. Only you can decide what’s right for you.
http://gutcritters.com/dietary-opioid-peptides-antioxidant-status-and-dna-methylation/As those of you who have read this blog for any length of time know, I’m not a big fan of foods that form opioid peptides upon digestion. As I explained in part two of my GERD series, all exogenous opioids, whether derived from prescription medications, illicit drugs, or food, slow and in severe cases stop both gastric and intestinal movement.
In the stomach, reducing the frequency by which food is released into the small intestine can result in acid reflux. The longer food sits in the stomach, the more prone it is to work its way back into the esophagus, especially if swollen by insoluble fiber.
In the intestine, inhibition of the wave-like muscle movements that propel remnants of digested food to its final destination is a major predisposing factor for the development of small intestinal fungal and bacterial overgrowth (SIFBO). It is these peristaltic movements, and not the ileocecal valve at the boundary of the small intestine and colon, that is mainly responsible for preventing bacteria from the colon, especially the gram-negative variety, from working their way into the small intestine. It is for this same reason that thyroid disorders that depress metabolic rate like Hashimoto’s thyroiditis and euthyroid sick syndrome predispose to developing SIFBO. (1)
But opioids are best known for their pain-suppressing, and not gut-paralyzing, effects. It is this very quality that makes them so effective as analgesics.
There has been some debate about how pronounced these actions really are when it comes to opioid peptides (exorphins) formed from digesting A1 casein dairy and gluten grains. Given the studies I cited in my GERD post, I believe the evidence is strong enough to warrant caution.
Further evidence for these effects was inadvertently provided by a dietary study published earlier this year.
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I need to emphasize that these dairy opioids are only formed from digestion of A1 casein dairy, not from A2 casein sources like goat’s milk, milk from Jersey cows, or milk from Asian and African breeds. So while I stand by my belief that dairy does an intestinal tract good (at least in those without lactose intolerance), it’s mainly true in regards to A2 casein milk.
However, when it comes to gluten grains, there are no compensatory components of these foods to counteract their gliadin-derived opioid peptides. Whereas A1 dairy forms just one opioid type upon digestion, gluten has been shown to form many. These include gluten exorphins A5, A4, B5, B4, and C5. (21) (22)
Addition of alpha gliadin 7 to this list gives us at least six different opioid peptides formed upon digestion of gluten grains, all with the potential to negatively affect cysteine uptake, antioxidant status, and DNA methylation. This no doubt explains why the women I wrote about in this post regained their health on a gluten-free diet, but didn’t experience any relief eliminating dairy.
Combining A-1 dairy and gluten opioids, as would normally occur when eating a wheat-based breakfast cereal for example, would suggest that in the real-world, the effect on cysteine uptake would be cumulatively worse.
But this is far from the only issue with gluten grains. As I wrote in this post, these foods, especially in their whole-grain form, are rife with anti-nutrients and natural pesticides, chief among the latter being wheat germ agglutinin. They also contain phytic acid which prevents the proper absorption of the minerals they contain unless first soaked in an acidic medium for a specific length of time.
These grains also contain amylase and trypsin inhibitors. Amylase is an enzyme necessary for the proper digestion of starches, while trypsin is needed to properly digest protein. These inhibitors have been shown to cause the release of pro-inflammatory cytokines via the same receptors lipopolysaccharides (LPSs) trigger to activate the immune system. (23)
All these factors explain why there is not a lick of scientific evidence, outside of confounder-prone nutritional epidemiology, that consuming these grains are healthy. That is made abundantly clear in Anthony Colpo’s book: Whole Grains, Empty Promises: The Surprising Truth about the World’s Most Overrated ‘Health’ Food.
Those who believe that these issues are confined to modern gluten grains, and can be avoided by eating more ancient wheat varieties, are sadly mistaken. These ancient grains have an even higher prolamine-rich content than modern wheat.
A research paper that examined two ancient wheat strains–Graziella Ra and Kamut–found no evidence that they were any less toxic to celiacs than modern wheat. (24) And the reason is because we DO NOT produce either the pancreatic or brush-border enzymes necessary to break these prolamine bonds.
The 33-mer peptide that is the trigger for celiac disease in the genetically susceptible remains intact after fifteen hours of exposure to human digestive enzymes. (25) If that particular peptide is so resistant to digestion, what hope do any of us have when it comes to gliadin opioid peptides?
That said, there is some evidence to suggest that in an intestine with healthy gut flora, the pro-inflammatory effect of these grains may be somewhat mitigated. Bifidobacterium bifidus (B. bifidus), for example, has been shown to counter the inflammatory effects of gluten on cells lining the gut wall. (26) And as I wrote in this post, addition of prebiotics to pasta countered the negative effects of gluten on intestinal, tight-junction proteins.
Nonetheless, given the wide prevalence of autoimmune disorders, allergies, metabolic syndrome, anxiety, depression, chronic fatigue syndrome, fibromyalgia, IBS, ulcerative colitis, Crohn’s disease, SIFBO, etc., etc., etc., I think we can safely dispense with the notion that most people are sporting healthy gut flora, especially in the age of antibiotic overuse. On the contrary, all signs point to increasing levels of gut dysbiosis.
http://gutcritters.com/to-dairy-or-not-to-dairy-that-is-the-question/Once that happens, it’s the poor, over-worked liver that receives the full brunt of this antigen load. How well it handles this assault and whether those antigens enter systemic circulation depends on how bad the endotoxemia is and the overall health of this organ.
Not all claudins maintain intestinal integrity, however. Some claudins seal the gut (are pore-sealing), while others make it more permeable (are pore-forming).
Claudins -1, -3, -4, -5 and -8 have been classified as pore-sealing proteins. You want these claudins up-regulated along the length of your digestive tract to ensure a tight seal against the contents of your gut.
Claudin-2, however, is a pore-forming junction protein. The expression of this claudin increases intestinal permeability.
In colonic biopsies performed on patients with Crohn’s disease, claudin-2 is strongly elevated. Meanwhile, pore-sealing claudins -5 and 8 are down-regulated in this disorder. (2)
Ulcerative colitis is another inflammatory bowel disease characterized by up-regulation of claudin-2. So too collagenous colitis where increased expression of this protein is associated with a low expression of pore-sealing claudin-4. (3)
It should surprise none of you that beneficial bacteria and their metabolites affect tight junction proteins through numerous mechanisms. Lactobacillus rhamnosus, Lactobacillus plantarum, Lactobacillus acidophilus, Bifidobacterium breve, Bifidobacterium bifidum, and Bifidobacterium infantis have all been found to maintain or strengthen gut-barrier function and protect against endotoxemia. (4) (5) (6) (7)
Food components can also affect tight-junction integrity as you’ve already learned by reading this blog. They can do so either directly by affecting zonulin expression or by encouraging the growth of gut pathogens. Some food constituents, like gluten, do both.
Returning to dairy—milk, whey protein, and whey protein concentrate—all contain high levels of transforming growth factor beta (TGFβ). TGFβ is a type of anti-inflammatory cytokine. This family includes a subfamily named transforming growth factor beta 1 (TGFβ1).
These proteins have important properties that inhibit the initial growth of cancer cells and induce their death or apoptosis. They are also involved in regulating immune function. TGFβ1 is found in highest levels in the whey fraction of human and cow’s milk. Breast milk naturally has a very high level of TGFβ1.
In an in vitro study published in 2011, whey protein concentrate rich in TGFβ1 increased the expression of pore-sealing claudin-4 without affecting claudins-1, -2, -5, -7 or occludin. (
By doing so, it increased intestinal barrier function.
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However, whey isn’t the only dairy protein under current study for its effects on the intestinal wall. A second study has also hinted at casein’s ability to strengthen gut-barrier function in a rodent model. (10) In this study, rats genetically bred to be susceptible to the development of autoimmune type 1 diabetes were placed on either a high-casein or standard plant-based diet.
The high-casein diet was a modified AIN-93G rodent chow and contained the following: 200 g/kg casein, L-cysteine, corn starch, sucrose, soy-bean oil, cellulose, mineral mix, vitamin mix, choline bitartrate and butylated hydroxyanisole antioxidant.
The ingredients in the plant-based chow were: wheat, meat meal (80% sterilized), yellow dent corn, whole oats, wheat middling’s, alfalfa, soya oil, dried yeast, dicalcium phosphate, calcium carbonate, NaCl, dl-methionine, vitamins and trace elements.
Do be sure to note that wheat was the number-one ingredient in the plant-based chow and that sugar (sucrose) was not a constituent of this diet.
Levels of lactulose and mannitol were measured in the urine of these rats to detect intestinal permeability. Animals on the high-casein (HC) diet had lower levels of intestinal permeability than their cohorts. Serum zonulin levels, a marker for intestinal permeability, were also lower in the rats fed the high-casein diet.
Rats on the HC diet were found to have increased levels of pore-sealing claudin-1 and reduced expression of pore-forming claudin-2. Because of this, the incidence of autoimmune type 1 diabetes in the high-casein rats was 50% less than in the gluten-fed animals.
The differential effect seen in this study has several possible explanations.
A high-casein diet has been seen to increase mucin levels (mucus) in the guts of diabetes-prone rats. (11) Doing so decreased their intestinal permeability and lowered insulin levels. The mucosal layer is an important physical part of our gut barrier and along with beneficial bacteria, prevents a “leaky gut”.
Another finding was that the high-casein diet increased levels of two anti-inflammatory cytokines: interleukin 10 (IL-10) and the above-mentioned transforming growth factor beta 1. By doing so, the HC diet suppressed inflammation in these rodents which would have inhibited intestinal permeability.
Finally, the presence of gluten in the plant-based rodent chow had obvious negative impacts on the tight junctions of those animals.
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The formation of opioid peptides from consuming A-1 beta-casein dairy must be taken into consideration in cases of autism-spectrum disorders, constipation and gut dysbiosis as I’ve mentioned before. It would be wise, therefore, to seek out A2 beta-casein dairy while healing and sealing the gut. Once dysbiosis is a distant memory, the consumption of even A1 beta-casein is likely to not pose a problem for most.
Disentangling whether gluten or dairy proteins are causing health problems is a major confounder in observational studies and anecdotal accounts implicating dairy. The reality is that the consumption of dairy and gluten-grains very often goes together: cheese and crackers, mac and cheese, cookies and milk, cake and ice cream, beer and nachos, grilled cheese sandwiches, pizza, wheat cereal and milk, butter and toast, cheeseburger and bun, etc., etc., etc.
I would hypothesize that the high dairy consumption in the West is probably protective against the permeability-enhancing effects of gluten. For example, ricotta, a cheese high in whey protein and a common ingredient of lasagna, may make that dish far healthier than would otherwise be the case.
Pointing out that Asian cultures are not big consumers of dairy and that this somehow “proves” it’s hazardous to health holds very little water with me. The reality is that the traditional grain eaten in large parts of Asia is rice, a grain that does not have known negative effects on gut-barrier function.
This difference may be largely accountable for the variance in health outcomes between Eastern and Western populations. Unfortunately, those differences are rapidly disappearing due to the increasing substitution of wheat for rice as the staple grain of these societies.
The point he is missing above is that for as long as our cows are fed GMO corn, pesticides, growth hormones, and the milk is pasteurized, homogenized, then meat and dairy products are poison!http://gutcritters.com/small-intestinal-bacterial-overgrowth-part-five-dietary-causes-of-gastric-barrier-dysfunction-and-gerd/So here’s my recipe for the ultimate GERD experience. Consume a large meal containing lots of whole-wheat, add some beans, some very spicy tomato-salsa, perhaps a chunk or two of A1 beta-casein containing cheese, wash it all down with copious quantities of alcohol and immediately rush off to bed. In no time, you’ll be uttering those poetic words of endearment every spouse wants to hear: Honey, my acid reflux just stained the sheets. Magic, absolute magic!
http://gutcritters.com/risk-factors-small-intestinal-bacterial-fungal-overgrowth/After their endoscopy and insertion of gastrointestinal sensors, a meal was fed to all participants before they were sent home. This meal consisted of the following: a chicken sandwich, 6 oz. glass of milk, a cookie, and a banana.
Anyone see a problem with this hospital feast?
The sandwich bread, cookie, and likely milk would all have produced opioid peptides upon their digestion. Recall that humans lack the digestive enzymes to break proline bonds. While there is some speculation that certain species of beneficial gut flora may be able to do just that, the last thing in the world most of these patients could claim to have are healthy colonies of gut flora.
Eating both the sandwich bread and cookie would lead to the formation of various gluten opioid peptides. The milk served was likely from an A1 beta casein dairy source as that is the typical dairy type consumed in the United States. Ingesting it would have formed beta-casomorphin 7. And of course what these peptides do to antioxidant status via inhibition of cysteine uptake would also likely negatively affect motility via up-regulation of inflammatory cellular processes that would maintain an environment hospitable to pathogens.
No indication was given regarding what else was eaten by these participants once they left the medical center. However, given that the study was conducted at the University of Iowa in Iowa City, IA, I’ll leave it to your fervid imaginations for what other gluten and dairy opioid forming delicacies were ingested during the off-site monitoring period.
As ALL opioids slow gastrointestinal function, the conclusions drawn from this motility study are inherently confounded. We have no idea how much of this dysmotility can be blamed on gut dysbiosis as opposed to diet.
Conclusion
Again I reiterate that flinging open the gastric-barrier door by taking PPIs or impairing intestinal peristalsis predisposes to small bowel overgrowth whether by bacterial or fungal organisms or both.
Powerful drugs always have powerful side effects, and PPIs are no exception. For more on what those side effects are, I again refer you to this post.
As for delayed transit time, evidence is robust that the more often peristalsis is inhibited, the more likely a person is to suffer from recurring small bowel infections. Diet is surely a factor with both opioid peptides and binge drinking high on the list, but it’s not the only one.
Opioid drug use and thyroid disorders like Hashimoto’s thyroiditis or euthyroid sick syndrome (ESS) are also contributors. But even here, ESS is related to diet as it’s often caused by either prolonged fasting or chronic ketogenic dieting.
http://gutcritters.com/fibromyalgia-irritable-bowel-syndrome-and-endotoxemia/I strongly caution against the use of non-steroidal anti-inflammatory drugs (NSAIDs) like ibuprofen to treat your pain and discomfort. I fully recognize that doing so may aggravate your symptoms. However, the well-documented negative effect these types of analgesics have on gut barrier function is the last thing you need when confronting a severe case of endotoxemia.
A survey of fibromyalgia patients rated NSAIDs more effective than acetaminophen in relieving their pain and hinted at their widespread use in this population. (7) The long-term consequences of using these drugs, however, are clearly self-defeating when intestinal “leakiness” is suspected.
While the use of non-NSAID tramadol has been found effective in managing pain, reports of its constipating side-effects concern me greatly. This isn’t surprising as this drug works on opioid receptors, including those in the gut.
Constipation exacerbates endotoxemia. Peristalsis not only propels partially digested food and feces along, it also serves a vitally important housekeeping function by preventing pathogens from attaching to and breaching the gut wall. Once peristalsis is compromised, the risk of gram-negative bacteria entering the portal vein to the liver and translocating to systemic circulation increases.
Also recall that impaired peristalsis is the number one reason colonic gram-negative pathogens are able to migrate to the small intestine. Opioids, including those formed by the partial digestion of gluten or A1 beta-casein, always impact peristalsis for the worse.
The dysbiosis at the heart of fibromyalgia, not to mention IBS, cannot be overcome without clearing up the infection and repopulating the gut with beneficial flora. Anything less will guarantee the persistence of these conditions.