If my data are cherry picked, then where are all the data showing that vaccines don't work?
SARS-CoV-2 has non-human reservoirs and is now endemic. It will never go away. Period. It cannot be eradicated by vaccination, and anyone telling you it can be straight-up eradicated like smallpox with a high level of vaccination is lying. The current agenda of boosters will lead, inevitably, to bi-yearly shots, because the vaccine antibodies are waning within 6 months.
https://www.medicalnewstoday.com/articles/waning-immunity-and-covid-19-vaccines-how-worried-should-we-behttps://www.realclearscience.com/articles/2021/08/23/lets_stop_pretending_about_the_covid-19_vaccines_791050.htmlThe rate of breakthrough cases is much, much higher than we are being told, because public health agencies are engaging in deliberately lax surveillance for them to make the vaccine appear more effective than it really is.
https://fee.org/articles/what-is-the-true-vaccine-breakthrough-rate-the-cdc-doesnt-want-you-to-know/https://www.cbsnews.com/news/new-covid-19-cases-united-states-almost-all-among-people-unvaccinated/There is a preprint paper indicating that current SARS-CoV-2 strains are about to completely escape antibodies produced by these vaccines, and that the antibodies may even become non-neutralizing and infection-enhancing:
https://www.biorxiv.org/content/10.1101/2021.08.22.457114v1Shi Zhengli's own work indicates that betacoronaviruses such as SARS and MERS may have Dengue-like ADE:
https://journals.asm.org/doi/10.1128/JVI.02015-19I tried raising the alarm on this back in February of 2020 and it went mostly ignored.
Vaccines failing due to inducing immune sensitization has a historical precedent in the recent past. Sanofi's Dengvaxia vaccine failed because it triggered enhanced disease.
https://jeffreydachmd.com/2021/08/director-of-cdc-rochelle-walensky-warns-of-ade-antibody-dependent-enhancement-from-israel-data/https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8427162/The MATH+ protocol is written by a scan organization, provides no data to support their recommendations, and is headed by individuals with vested interests in the fake treatments they recommend. Looking at the treatments they and you are suggesting:
Vitamin C:
There are no controlled trials that have definitively demonstrated a clinical benefit for vitamin C in critically ill patients with COVID-19, and the available observational data are inconclusive.
People with COVID-19 hyperinflammation are suffering from a form of sepsis.
https://www.healthleadersmedia.com/clinical-care/expert-severe-covid-19-illness-viral-sepsishttps://pubmed.ncbi.nlm.nih.gov/34590796/It has previously been suggested that antioxidants are useful for treating sepsis, because sepsis involves the over-activity of pro-oxidant enzymes that the body uses to fight infection.
https://journals.lww.com/ccmjournal/Abstract/2007/09001/Antioxidant_supplementation_in_sepsis_and_systemic.25.aspxIt is entirely possible that COVID-19 hyperinflammation is, much like Keshan disease, a disease of low antioxidant capacity (redox equilibrium issues in the body due to chronic oxidative stress, i.e., endothelial dysfunction) that can be counteracted by raising levels of antioxidant substrates, such as selenium, glutathione, et cetera.
Many people who died of COVID-19 had issues pointing towards low antioxidant capacity. Low Vitamin D, low glutathione, low selenium, and so on.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7385774/https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7937041/https://pubmed.ncbi.nlm.nih.gov/32463221/This points to an inescapable conclusion; COVID-19 causes death by aggressive lipid peroxidation brought on by sepsis.
Though antioxidants seem like a logical counter to this (in South Korea, they tested the NADPH oxidase inhibitor APX-115 against the virus), there is no guarantee that they will have bioavailability to the affected cells. I'm not completely gung-ho about it; I understand that there are serious challenges involved here, and that what looks promising on paper may not always improve a patient's condition.
People are hunting for a magic bullet for COVID-19, but no such magic bullet exists. In my opinion, the best prophylaxis against COVID-19 is a balanced, micronutrient-rich diet, strenuous exercise, and maintaining a healthy BMI, because this induces physiological changes in the vasculature that make them more resilient (it raises nitric oxide levels and antioxidant capacity and reduces oxidative stress). This is reflected by evidence. COVID-19's severity is GREATLY enhanced with increasing body mass index.
https://www.sciencedirect.com/science/article/abs/pii/S1262363620300975?via%3DihubThis isn't just a get-healthy-quick pill. It's a lifestyle change. It's also why I recommend against lockdowns, and perhaps for more aggressive distancing (as in 15+ feet) to compensate. A good, solid crosswind will blow any aerosols away, and UV from direct sunlight will degrade and destroy the virions. The greatest risk of transmission is mostly indoors, in tightly-packed rooms with stagnant air.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7673425/https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8305330/People should be outdoors, they should be jogging and biking, and they should be maintaining a high activity level. Becoming sedentary makes COVID-19 infections way, way worse. It turns the blood vessels into a buffet table for the virus.
That's disappointing, if true. I have been in contact with pharmacology experts who claimed anecdotal evidence for fluvoxamine improving patients' conditions, but there do have to be well-designed clinical trials to show whether or not it has any benefit. There are many plausible mechanisms by which fluvoxamine may improve a patient's condition.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8094534/Budesonide does have some evidence, but it is inferior to IV dexamethasone which we have been using for all our inpatients for months.
Again, disappointing. Also, a little odd, given how promising it seemed before.
https://pubmed.ncbi.nlm.nih.gov/33844996/https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8310374/The links in your original letter consider these drugs in the context of rodent peptic ulcers, among other things. Hardly applicable to humans with COVID.
Not necessarily true. Those papers describe a mechanism. While I would never argue that rodent gastric cells are the same thing as human airway or vascular epithelial or endothelial cells, there are many features that are homogenous between different eukaryotic cell lines, and an effect demonstrated in one type of cell might also be applicable to another. It's not conclusive proof of therapeutic effect, but it does point towards a fresh avenue of study.
The problem I have is that these drugs aren't really being studied as aggressively as they should be. There should be more trials of antioxidants, not just one or two here and there. They keep pushing antivirals that don't seem to work on hyperinflammatory COVID-19, when, at the very least, antioxidants have a plausible therapeutic mechanism. You'll never know if you never look.
I don't think any one drug will treat this on its own, but perhaps a cocktail of these may provide a marginal benefit. The trouble is that hyperinflammatory COVID-19 is a real son of a bitch.
There's some truth in this, but not to degree you state. If there was enough hypochlorous acid to destroy hemoglobin to such a degree, why do we not see a severe hyperchloremic acidosis?
See the following:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7757048/https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3222650/This would be a logical result of neutrophilia and NETosis in COVID-19. Let's review the papers on that for a moment.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8376580/https://www.sciencedirect.com/science/article/pii/S221249262030052XOkay, so there are lots of NETs around. More than macrophages can reasonably be expected to clean up in a reasonable time frame. So, there is going to be a lot of extracellular myeloperoxidase lying around, making hypochlorous acid from hydrogen peroxide and chloride ions.
This would, necessarily, lead to the liberation of iron and tons of Fenton reagent lying around.
https://pubmed.ncbi.nlm.nih.gov/33974898/https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7836924/Though metabolic and respiratory alkalosis are more common, some critically-ill COVID-19 patients who died did indeed have severe acidosis.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7236721/https://pubmed.ncbi.nlm.nih.gov/33103442/COVID-19 causes glucose and lipid-handling issues, as well.
https://journals.plos.org/plospathogens/article?id=10.1371/journal.ppat.1009243https://pubmed.ncbi.nlm.nih.gov/33043283/https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7570435/The metabolic shifts are rather profound.
Why do we not see profound anemia due to hemoglobin destruction?
Decreased serum hemoglobin and increased serum ferritin have been found in COVID-19:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7753740/https://casereports.bmj.com/content/13/12/e238118https://link.springer.com/article/10.1007%2Fs10654-020-00678-5https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7267810/Why does intubation and proning make the SpO2 go from 70% to 95%? Why does it make the PaO2 go from 60 mm Hg to 120 mm Hg?
Trying to claim that intubation and ventilation is actively killing people is just plain incorrect.
I said, in the letter, that it was a catch-22, because you need oxygen to live. Intubation does increase oxygenation, but also causes VILI-like damage and additional oxidative stress alongside that.
It's not
just a blood problem. The blood-air barrier itself and gas exchange across it are also compromised due to the endothelial injury itself:
https://www.tandfonline.com/doi/abs/10.1080/21688370.2021.1937013?journalCode=ktib20ARDS/Acute Lung Injury involves a great deal of neutrophilia in the pulmonary vasculature and oxidative damage to the tissues.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6801694/OK, so show me the randomized control trials which show ivermectin is an effective prophylactic. There is currently no evidence for this either:
Owing to very low certainty of evidence, the effect of ivermectin, compared with standard care, in reducing the risk of suspected, probable, or laboratory confirmed infection remains very uncertain; the certainty of evidence was rated down because of serious risk of bias and very serious imprecision.
I'm happy consider any treatment provided there is some evidence it works. You have failed to provide that evidence.
See for yourself who they enrolled for all the RCTs the media is citing as proof of the ineffectiveness of antivirals, like Oxford's completely botched RECOVERY study.
https://covexit.com/recovery-covid-19-research-blasted-for-toxic-dosage-towards-oxfordgate/https://www.ox.ac.uk/news/2020-06-05-no-clinical-benefit-use-hydroxychloroquine-hospitalised-patients-covid-19These studies virtually all enroll people who have COVID-19 hyperinflammation, have been symptomatic for around a week or more, and have been hospitalized. I have already explained why this is futile. The virus is gone. And I mean, it's
gone. It's too late.
https://www.mdpi.com/1999-4915/13/6/963/htmAn article written by a computer scientist and a practitioner of "energy medicine", published in a fake journal which is not listed in PubMed, has no publisher, has literally zero impact factor, which is ran by a young Earth creationist and a lawyer who sues vaccine companies on behalf of "victims". And even then, they still state "there are no studies demonstrating definitively that this is happening".
Come on. You seem smart enough to realize just how disingenuous and dishonest something like this is.
Very well, then. I shall go over my concerns in brief, citing my own letter (which, in turn, cites many other files).
https://mega.nz/file/HZNmyRKB#xF15FrsAEZkwBPi4tdUP5toBBqeRHDJJAHzZt6Hg_Qg-The vaccine is not sterilizing and does not prevent transmission. The vaccinated are still contagious to others. This means that the virus no longer has any pressure to become less virulent; mutations that could be lethal to the unvaccinated may only cause a mild increase in illness in the vaccinated.
-Natural immunity from a prior infection results in antibodies to all of the virus's proteins, not just one.
-All of the current COVID-19 vaccines have undergone highly accelerated trial periods, not allowing any time for long-term side effects to appear.
-Messenger RNA vaccines, which deliver the active form of genes to cells to synthesize viral proteins and produce an antigen response that way, have never been tested in humans before. In Moderna's case, mRNA-1273 is actually their first-ever commercial product. Would you willingly put a company's first-ever product in your body?
-The production and validation of these vaccines involved fetal cell lines, which some may object to.
-The lipid nanoparticles from the mRNA vaccines have been shown to bioaccumulate all over the body, and do not stay in the shoulder.
-The PEGylated lipid nanoparticles in mRNA vaccines can occasionally trigger severe allergic reactions.
-Damaged mRNA can stall ribosomes by getting jammed in them, causing ribosome attrition and reduced protein synthesis.
-The method of making SARS-CoV-2 Spike inert, inserting prolines on the S1/S2 boundary to rigidly lock the trimers in the prefusion conformation, does not take into account any unexpected proteolysis or further processing of the Spike proteins by the body that may release the S1 subunits, allowing them to travel freely around the body and bind to things.
-SARS-CoV-2 Spike is, in itself, a pathogenic protein, capable of binding to ACE2, integrins, neuropilin-1, and bacterial LPS. It can induce autoantibody responses against healthy tissue, overactivate T-cells with a SAg region, penetrate the blood-brain barrier, and bind to heparin-binding proteins and induce amyloid aggregation and possible neurodegeneration, among many other unknown and possibly pathogenic effects.
-SARS-CoV-2 may have ADE, which means that a future strain may cause antibodies from vaccines based on the sequence of previous strains to become non-neutralizing, turning them into trojan horses that help virions infect leukocytes they would not have otherwise been able to infect.
-Messenger RNA that codes for Modified SARS-CoV-2 Spike may be integrated into the genome of cells by endogenous LINE-1 reverse transcription. There is a plausible mechanism whereby it can integrate itself into your DNA. That means that this is, in fact, potentially a gene delivery system.
Out of all of these, the most appalling thing is this:
https://pubmed.ncbi.nlm.nih.gov/33328624/And the rest of your links go the same way as this one, and the same way as your original letter - off the deep end of conspiracy theories with absolutely no supporting evidence or facts.
You said there was no proof that mind-controlling nanoparticles existed. I showed you that not only do they exist, and not only did James Giordano give presentations about them before an entire class of stunned cadets at West Point, they are described explicitly in publicly-available materials.
Not only that, DARPA, DTRA, and vaccine researchers are intimately connected both to brain-computer interface research, and to GOF research at the Wuhan Institute of Virology.
The degrees of separation here are minuscule. When plotted out on a node graph, they would all cluster together. David Martin and M-CAM showed, beyond a shadow of a doubt, that GOF SARS strains and their features are basically patented products.
That's not a conspiracy theory. That is a RICO case the size of Mount Everest. It is also mass murder and treason.
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